The above symptoms are not temporary. The patient's head may feel dizzy for several hours in a row, and nystagmus does not disappear for several days. After the end of the attack, the patient has a unsteady gait for a couple of weeks.
It is important for the diagnosis of pathology to collect the most complete anamnesis of life and disease. It is also worth noting that an important fact for making a diagnosis is that with this pathology, unlike other ailments with similar symptoms, there is no impairment of auditory function.
At present, there are special neurological tests that make it possible to comprehensively assess the functioning of the human vestibular apparatus. These include:
Treatment of vestibular neuritis is aimed at eliminating the cause of the progression of the pathology and at reducing the manifestation of symptoms. For this purpose, the patient is prescribed pharmaceuticals from the group of suppressants.
The drug of choice is dimenhydrinate. The following medications are also prescribed:
Treatment of pathology is carried out in stationary conditions under the supervision of tadacip specialists. If necessary, the treatment plan can be adjusted. A good effect in the treatment of the disease is given by therapeutic exercises and physiotherapy.
Vestibular neuronitis as a disease was noted at the beginning of the 20th century by Eric Ruttin, but a more detailed description was given in 1924 by Carl Nilen, when some diagnostic points were also described. The term itself was introduced by Charles Hallpike in the middle of tadacip pills.
The disease is statistically the third most common cause of acute systemic (vestibular) dizziness. Benign paroxysmal positional vertigo (a commonly used abbreviation for BPPV) ranks first, followed by Meniere's disease.
It is noted equally in men and women, most often occurs at a relatively young (28-58 years) age.
Information for doctors. The diagnosis code according to the ICD-10 classification H81.2 refers to ENT diseases, however, often patients with vestibular neuronitis end up in the primary vascular departments with suspected acute cerebrovascular accident.
The reasons for the development of vestibular neuronitis are unknown. According to the most common theory, inflammation of the infectious-allergic or viral nature of the vestibular nerve takes place. In favor of the presence of a virus (more often type I herpes virus, as well as a new coronavirus infection), such features as the frequent development of neuronitis after a respiratory viral infection, the presence of epidemiological outbreaks in late spring, as well as noted cases of familial development of neuronitis (when the carriers of the infectious agent were one member of the family.Specifically, the herpes virus is a clinical case of the development of herpetic encephalitis in this disease.
